By V. Mannig.
Holly has been diagnosed with Dissociative Identity Disorder for a few years now generic acarbose 50 mg without prescription diabetes diet snacks. She finally came to terms with the diagnosis and is now trying to bring public awareness to this illness generic 25 mg acarbose amex diabetic low blood sugar. Plus some of the misconceptions people have about those who live with Dissociative Identity Disorder (DID) generic acarbose 50mg with mastercard blood glucose blood test. If you find this Dissociative Identity Disorder video helpful, please share it with others through the Facebook "Like" button or bookmark share button at the bottom of the page. We invite you to call our automated number at 1-888-883-8045 and share your experience in living with DID. Her earliest recollection of living with a multiple personality was at 4 years old. At one time in her young life, she was coping with as many as 58 personalities. Now a mother of three in her fifties, Maria has managed to cope with her personalities and has some advice she would like to share with others. Childhood sexual abuse video interview on the long-term effects of being sexually abused as a child. Our guest shares her experiences of childhood sexual abuse and developing DID (Dissociative Identity Disorder) as a way to cope. Child sexual abuse can bring very serious long-term consequences such as Dissociative Identity Disorder (DID), formerly known as Multiple Personality Disorder. DID can be a coping method after being exposed to a very severe trauma. Maggie Claire, our guest on the HealthyPlace Mental Health TV Show, talks about the long-term impact of being sexually abused as a child. We invite you to call our number at 1-888-883-8045 and share your experience in dealing with child sexual abuse and dissociative identity disorder. Even after the abuse has ended, the traumatic memories remain. This conference focuses on how to effectively deal with those traumatic memories. Karen Engebretsen-Larash, psychologist and specialist in treating trauma-related disorders. Karen: Traumatic memories are any recollections either in the mind or body that the unconscious tries to communicate with the person who has been traumatized. These memories can occur at any time, even long after the sexual abuse has taken place. David: Why is it that long after experiencing sexual abuse, some people are left with very vivid traumatic sexual abuse memories that are difficult to deal with, much less get rid of? Karen: The mind has a way of protecting itself from pending danger and does a pretty good job at protecting the self; but in times of great stress, it is likely for these memories of sexual abuse to increase in frequency which is a signal that the unconscious can no longer continue to suppress this information. David: Some people say they are "haunted" by memories of traumatic experiences which intrude on and disrupt their daily lives. How can an individual deal with this in an effective manner? Karen: They can, but it generally takes years to work through the aftermath of repeated sexual trauma. He developed this technique to remove the "pain" aspect or the "self" figure so that patients can continue doing the uncovering work necessary for healing. Although his focus has been on the inpatient population, he has been making this available on an outpatient basis. In my clinical experience, I am amazed by how much more quickly we can speed up the therapy process following the Incorporation Therapy. David: Why do some people undergoing extreme stress have continuous memory and others have amnesia for all or part of their experience? We are all born with certain coping strategies and we learn at a very early age what is safe to let others know about us and what is not. Individuals who have "continuous" memories are generally so crippled that they cannot function. Others become extremely creative and develop a system whereby they can access different "parts" (or alters) to cope with the stressful situations. This is the extreme form of PTSD (post-traumatic stress disorder) and can lead to Dissociative Identity Disorder (DID). Certain things can trigger a memory that may not have bothered you in the past. David: If you can remember the abuse but not the feelings associated with them, only visual memories, how do you get in touch with those feelings? It is likely to believe that you were told that you were not permitted to feel in any way shape or form.
For comprehensive information on obsessive-compulsive and other types of anxiety disorders generic acarbose 50 mg amex signs of diabetes in young dogs, visit the Anxiety-Panic Community discount 25 mg acarbose mastercard diabetes type 2 uk statistics. Diagnostic and Statistical Manual of Mental Disorders order acarbose 25 mg mastercard blood glucose units of measurement, Fourth Edition. Merck Manual, Home Edition for Patients and Caregivers, last revised 2006. Full description of Obsessive-Compulsive Personality Disorder (OCPD). Definition, signs, symptoms, causes of Obsessive-Compulsive Personality Disorder. About 16 million Americans, or 8-percent of the population have Obsessive-Compulsive Personality Disorder, making it one of the most prevalent personality disorders in the U. Rigid adherence to rules and regulations and an overwhelming need for order and personal control are the primary characteristics of obsessive compulsive personality disorder. People living with OCPD are inflexible, perfectionists and unwilling to yield responsibilities to others. They are reliable, dependable, orderly, and methodical, but their inflexibility makes them unable to adapt to change. Because they are cautious and weigh all aspects of a problem, they have difficulty making decisions. According to the Merck Manual, "people with an obsessive-compulsive personality are often high achievers, especially in the sciences and other intellectually demanding fields that require order and attention to detail. However, their responsibilities make them so anxious that they can rarely enjoy their successes. Many people confuse Obsessive-Compulsive Personality Disorder (OCPD) with Obsessive-Compulsive Disorder (OCD). People with OCD are often aware that their obsessions are abnormal, but are compelled to perform them anyway. People with obsessive compulsive personality disorder, however, believe their need for strict order and rules is perfectly normal. A pervasive pattern of preoccupation with orderliness, perfectionism, and mental and interpersonal control, at the expense of flexibility, openness, and efficiency, beginning by early adulthood and present in a variety of contexts, as indicated by four (or more) of the following:is preoccupied with details, rules, lists, order, organization, or schedules to the extent that the major point of the activity is lostshows perfectionism that interferes with task completion (e. Parenting styles and family dynamics may also play a role in the development of OCPD. One theory suggests that as children, people with Obsessive-Compulsive Personality Disorder were consistently punished for negative behavior, failure, and rule-breaking, while receiving no praise for success and compliance. To avoid punishment, the child develops a habit of rigidly following rules that lasts into adulthood. For comprehensive information on obsessive-compulsive and other personality disorders, visit the Personality Disorders Community. Full description of Oppositional Defiant Disorder (ODD). Definition, signs, symptoms, causes of Oppositional Defiant Disorder. All kids, especially very young children, are defiant, express anger, or talk back at some time or another. However, when the behavior becomes increasingly hostile, is a serious concern for parents, teachers and other adults, starts affecting family, friends and school performance, and continues for a significant amount of time, the child may be diagnosed with Oppositional Defiance Disorder. Children with Oppositional Defiant Disorder are stubborn, difficult, and disobedient without being physically aggressive or actually violating the rights of others. The Merck Manual reports ODD usually develops by age 8. There are various statistics on the incidence of Oppositional Definance Disorder in the U. Numbers range from 2-16% of children and teens in the U. Typical behaviors of children with Oppositional Defiant Disorder include arguing with adults; losing their temper; actively defying rules and instructions; blaming others for their own mistakes; and being angry, resentful, and easily annoyed. These children do know the difference between right and wrong and feel guilty if they do anything that is seriously wrong. The behaviors do not occur exclusively during the course of a Psychotic or Mood Disorder. Criteria are not met for Conduct Disorder, and, if the individual is age 18 years or older, criteria are not met for Antisocial Personality Disorder. Several theories about the causes of Oppositional Defiant Disorder are being investigated. Some studies also suggest that having a mother with a depressive disorder can result in a child with ODD. For more on oppositional defiant disorder and extensive information on parenting challenging children, visit the Parenting Community. Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition. Merck Manual, Home Edition for Patients and Caregivers, last revised 2006.
Dysthymia is a chronic form of depression discount 25 mg acarbose fast delivery diabetes test meters free, but is milder in severity than major depression acarbose 25mg low cost managing cat diabetes. People who suffer from dysthymia have a persistent underlying depression cheap acarbose 50mg on line metabolic disease epilepsy. Dysthymia (dysthymic disorder) usually sets in during early adulthood, and the disorder can last for years or even decades. Later onset is usually associated with bereavement or obvious stress, and often follows on the heels of a more extreme depressive episode. Women are twice as likely as men to suffer from dysthymia, similar to that seen with major depression. About 3% of the population is affected by dysthymic disorder, with three-quarters of individuals displaying signs of other psychiatric or medical disorders as well. Depressed mood for most of the day, for more days than not, as indicated either by subjective account or observation by others, for at least 2 years. Note: In children and adolescents, mood can be irritable and duration must be at least 1 year. Presence, while depressed, of two (or more) of the following:poor appetite or overeatinginsomnia or hypersomniapoor concentration or difficulty making decisionsfeelings of hopelessnessC. During the 2-year period (1 year for children or adolescents) of the disturbance, the person has never been without the symptoms in Criteria A and B for more than 2 months at a time. No Major Depressive Episode has been present during the first 2 years of the disturbance (1 year for children and adolescents); i. Note: There may have been a previous Major Depressive Episode provided there was a full remission (no significant signs or symptoms for 2 months) before development of the Dysthymic Disorder. In addition, after the initial 2 years (1 year in children or adolescents) of Dysthymic Disorder, there may be superimposed episodes of Major Depressive Disorder, in which case both diagnoses may be given when the criteria are met for a Major Depressive Episode. There has never been a Manic Episode, a Mixed Episode, or a Hypomanic Episode, and criteria have never been met for Cyclothymic Disorder. The disturbance does not occur exclusively during the course of a chronic Psychotic Disorder, such as Schizophrenia or Delusional Disorder. The symptoms are not due to the direct physiological effects of a substance (e. The symptoms cause clinically significant distress or impairment in social, occupational, or other important areas of functioning. This may increase the likelihood of developing later major depressive episodes. Major life stressors, chronic illness, medications, and relationship or work problems may also increase the chances of dysthymia. Genetics-heredity may also play a role in the development of dysthymia. Most people with dysthymia will have a family history of mood or affective disordersAlthough it can occur at any age, dysthymia often begins earlier than major depression - in childhood, adolescence, or early adult life. People who have dysthymia have been thought to have a "depressive personality" or "depressive neurosis. Definition, signs, symptoms, and causes of Histrionic Personality Disorder. Histrionic Personality Disorder is a condition in which a person acts very emotional and overly dramatic in order to call attention to themselves. People with this disorder are also intensely expressive, egocentric, highly reactive, and excitable. Other features of Histrionic Personality Disorder may include emotional and interpersonal superficiality as well as socially inappropriate interpersonal behavior. People with a histrionic personality are prone to sexually provocative behavior or to sexualizing nonsexual relationships. However, they may not really want a sexual relationship; rather, their seductive behavior often masks their wish to be dependent and protected. Some people with a histrionic personality also are hypochondriacal and exaggerate their physical problems to get the attention they need. A pervasive pattern of excessive emotionality and attention seeking, beginning by early adulthood and present in a variety of contexts, as indicated by five (or more) of the following:is uncomfortable in situations in which he or she is not the center of attentioninteraction with others is often characterized by inappropriate sexually seductive or provocative behaviordisplays rapidly shifting and shallow expression of emotionsconsistently uses physical appearance to draw attention to selfhas a style of speech that is excessively impressionistic and lacking in detailshows self-dramatization, theatricality, and exaggerated expression of emotionis suggestible, i. Psychoanalysts suggest that a traumatic childhood contributes towards the development of HPD. For comprehensive information on histrionic and other personality disorders, visit the Personality Disorders Community. Full description of major depression (clinical depression, major depressive disorder). Definition, signs, symptoms, and causes of major depression.
Of these patients purchase acarbose 25 mg with visa diabetic diet total carbs per day, 1004 participated in short-term placebo-controlled schizophrenia studies with doses of 20 mg buy acarbose 50 mg diabetes symptoms racing heart, 40 mg buy 50mg acarbose overnight delivery diabetes disability, 80 mg or 120 mg once daily. A total of 533 Latuda-treated patients had at least 24 weeks and 238 Latuda-treated patients had at least 52 weeks of exposure. Adverse events during exposure to study treatment were obtained by general inquiry and voluntarily reported adverse experiences, as well as results from physical examinations, vital signs, ECGs, weights and laboratory investigations. Adverse experiences were recorded by clinical investigators using their own terminology. In order to provide a meaningful estimate of the proportion of individuals experiencing adverse events, events were grouped in standardized categories using MedDRA terminology. The stated frequencies of adverse reactions represent the proportion of individuals who experienced at least once, a treatment-emergent adverse event of the type listed. Treatment-emergent adverse events were defined as adverse experiences, which started or worsened on or after the date of the first dose through seven days after study medication discontinuation. There was no attempt to use investigator causality assessments; i. It is important to emphasize that, although the reactions occurred during treatment with Latuda, they were not necessarily caused by it. The label should be read in its entirety to gain an understanding of the safety profile of Latuda. The figures in the tables and tabulations cannot be used to predict the incidence of side effects in the course of usual medical practice where patient characteristics and other factors differ from those that prevailed in the clinical studies. Similarly, the cited frequencies cannot be compared with figures obtained from other clinical investigations involving different treatment, uses and investigators. The cited figures, however, do provide the prescriber with some basis for estimating the relative contribution of drug and nondrug factors to the adverse reaction incidence in the population studied. The following findings are based on the short-term placebo-controlled premarketing studies for schizophrenia in which Latuda was administered at daily doses ranging from 20 to 120 mg (n = 1004). Commonly Observed Adverse Reactions: The most common adverse reactions (incidence ?-U 5% and at least twice the rate of placebo) in patients treated with Latuda were somnolence, akathisia, nausea, parkinsonism and agitation. Adverse Reactions Associated with Discontinuation of Treatment: A total of 9. There were no adverse reactions associated with discontinuation in subjects treated with Latuda that were at least 2% and at least twice the placebo rate. Adverse Reactions Occurring at an Incidence of 2% or More in Latuda-Treated Patients: Adverse reactions associated with the use of Latuda (incidence of 2% or greater, rounded to the nearest percent and Latuda incidence greater than placebo) that occurred during acute therapy (up to 6-weeks in patients with schizophrenia) are shown in Table 6. Table 6: Adverse Reaction in 2% or More of Latuda-Treated Patients and That Occurred at Greater Incidence than in the Placebo-Treated Patients in Short-term Schizophrenia StudiesNote: Figures rounded to the nearest integerBody System or Organ ClassDictionary-derived TermBased on the pooled data from the placebo-controlled, short-term, fixed-dose studies, among the adverse reactions that occurred with a greater than 5% incidence in the patients treated with Latuda, the apparent dose-related adverse reactions were akathisia and somnolence (Table 7). Table 7: Dose-Related Adverse EventsPercentage of Subjects Reporting Reaction* Somnolence includes adverse event terms: hypersomnia, hypersomnolence, sedation, and somnolenceIn the short-term, placebo-controlled schizophrenia studies, for Latuda-treated patients, the incidence of reported EPS-related events, excluding akathisia and restlessness, was 14. Akathisia appeared to be dose-related and the greatest frequency of parkinsonism and dystonia occurred with the highest dose of Latuda, 120 mg/day (Table 8). All EPS events, excluding Akathisia/RestlessnessIn the short-term, placebo-controlled schizophrenia studies, data was objectively collected on the Simpson Angus Rating Scale for extrapyramidal symptoms (EPS), the Barnes Akathisia Scale (for akathisia) and the Abnormal Involuntary Movement Scale (for dyskinesias). The mean change from baseline for Latuda-treated patients was comparable to placebo-treated patients, with the exception of the Barnes Akathisia Scale global score (Latuda, 0. The percentage of patients who shifted from normal to abnormal was greater in Latuda-treated patients versus placebo for the BAS (Latuda, 16. In the short-term, placebo-controlled clinical trials, dystonia occurred in 4. Laboratory Test Abnormalities and ECG Changes in Clinical StudiesLaboratory Test AbnormalitiesIn a between-group comparison of the pooled data from short-term, placebo-controlled studies, there were no clinically important changes in total cholesterol measurements; triglycerides or glucose from Baseline to Endpoint [see Warnings and Precautions (5. There were also no clinically important differences between Latuda and placebo in mean change from baseline to endpoint in routine hematology, urinalysis, or serum chemistry. Latuda was associated with a dose-related increase in prolactin concentration [see Warnings and Precautions (5. A creatinine shift from normal to high occurred in 3. The threshold for high creatinine value varied from ?-U 1. Transaminases: The mean changes in AST and ALT for Latuda- and placebo-treated patients were similar. The proportion of patients with transaminases (AST and ALT) elevations ?-U 3 times ULN was similar for all Latuda-treated patients (0. Electrocardiogram (ECG) measurements were taken at various time points during the Latuda clinical trial program.